(41) Dissociating self-generated from passively applied head motion: neural mechanisms in the vestibular nuclei.   Roy JE and Cullen KE.   2004.   Journal of Neuroscience, volume 24, pages 2102-2111.

(42) The cerebellar cognitive affective syndrome.   Schmahmann JD and Sherman JC.   1998.   Brain, volume 121, pages 561-579.   

(43) Mammalian play: training for the unexpected.   Spinka M, Newberry RC and Bekoff M.   2001.   The Quarterly Review of Biology, volume 76, pages 141-168.

(44) A PET study of voluntary movement in schizophrenic patients experiencing passivity phenomena (delusions of alien control).   Spence SA, Brooks DJ, Hirsch SR, Liddle PF, Meehan J and Grasby PM.   1997.   Brain, volume 120, pages 1997-2011.

(45) Functional and pathophysiological models of the basal ganglia.   Wichmann T and DeLong MR.   1996.   Current Opinion in Neurobiology, volume 6, pages 751-758.

(46) Correlation of primate caudate neural activity and saccade parameters in reward-oriented behaviour.   Itoh H, Nakahara H, Hikosaka O, Kawagoe R, Takikawa Y and Aihara K.   2003.   Journal of Neurophysiology, volume 89, pages 1774-1783.

(47) Behavioural disorders induced by external globus pallidus dysfunction in primates. 1. Behavioural study.   Grabli D, McCairn K, Hirsch EC, Agid Y, Feger J, Francois C and Tremblay L.   2004.   Brain, volume 127, pages 2039-2054.
      With reference to how researchers find what they are looking for, this study is devoid of any consideration of observer bias and of indices of observer reliability. The editor colluded.

(48) Context-dependent modulation of movement-related discharge in the primate globus pallidus.   Turner RS and Anderson ME.   2005.   Journal of Neuroscience, volume 25, pages 2965-2976.
     With reference to doubts conveyed by brackets and quotation marks, this article has many of both formats, sometimes in combination. The self-timed condition required observation of spontaneous movements to other locations, to control for the possibility that the movements under "memory-contingent" conditions were random and not based on memory.

(49) The behavioural and motor consequences of focal lesions of the basal ganglia in man.   Bhatia KP and Marsden CD.   1994.   Brain, volume 117, pages 859-876.

(50a) The substantia nigra of the human brain. 1. Nigrosomes and the nigral matrix, a compartmental organization based on calbindin D (28K) immunohistochemistry.   Damier P, Hirsch EC, Agid Y and Graybiel AM.    1999.   Brain, volume 122, pages 1421-1436.
     With reference to hyperbole, the difference to the groups was 60% to 40% in a sample of seven subjects with a possibility of post mortem changes, so calbindin immunostaining was shown to be a tool, not a "key tool". Calbindin D28k is expressed particularly in the cerebellum.

(50b) The substantia nigra of the human brain. 2. Patterns of loss of dopamine-containing neurons in Parkinson's disease.   Damier P, Hirsch EC, Agid Y and Graybiel AM.   1999.   Brain, volume 122, pages 1437-1448.
     With reference to making sense, how many diseased brains were studied? The sentence that begins "These zones were recognizable in all of the brains..." comes immediately after a precis of abstract 50a, which describes a study of seven normal brains.